![]() The lead authors are Andrew Yang, PhD, a postdoctoral scholar in Wyss-Coray’s group, and Fabian Kern, a graduate student in Keller’s group. Wyss-Coray shares senior authorship with Andreas Keller, PhD, chair of clinical bioinformatics at Saarland University. ![]() “And we couldn’t find it.”Ī paper describing the study will be published June 21 in Nature. “We used the same tools they’ve used - as well as other, more definitive ones - and really looked hard for the virus’s presence,” he said. Scientists disagree about whether SARS-CoV-2 is present in COVID-19 patients’ brains. “The brains of patients who died from severe COVID-19 showed profound molecular markers of inflammation, even though those patients didn’t have any reported clinical signs of neurological impairment,” said Wyss-Coray, who is the D. Brain samples from 14 people who died of other causes were used as controls for the study. ![]() Yet the researchers couldn’t find any signs of SARS-CoV-2 in brain tissue they obtained from eight individuals who died of the disease. About one-third of individuals hospitalized for COVID-19 report symptoms of fuzzy thinking, forgetfulness, difficulty concentrating and depression, said Tony Wyss-Coray, PhD, professor of neurology and neurological sciences at Stanford. And they can persist as an aspect of “long COVID,” a long-lasting disorder that sometimes arises following infection with SARS-CoV-2, the virus that causes COVID-19. These complaints increase with more severe cases of COVID-19. The findings may help explain why many COVID-19 patients report neurological problems. Investigators at the Stanford School of Medicine and Saarland University in Germany report that what they saw looks a lot like what’s observed in the brains of people who died of neurodegenerative conditions such as Alzheimer’s disease and Parkinson’s disease. ![]() The most comprehensive molecular study to date of the brains of people who died of COVID-19 turned up unmistakable signs of inflammation and impaired brain circuits. ![]()
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